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doi: 10.1371/journal.pone.0283389 91 LiuF.LiuY.LvX.LunH

Notably, the acetylation-defective mutant p53 3 KR, which cannot induce cell-cycle arrest, senescence or apoptosis, fully retains the ability to suppress SLC7A11 and trigger ferroptosis under oxidative stress (Le et al., 2015)

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It does not create an approved drug label, an FDA-approved indication, standardized national dosing, a guarantee of insurance coverage, or a finding that every formulation and route is appropriate

7A, B), suggesting both genes were regulated by DMF in an NRF2-dependent but not 5hmC-dependent regulatory manners