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IVIg directly influences the immune system through multiple complex mechanisms, including neutralizing pathogenic autoantibodies, modulating the expression of Fc receptors on immune cells, inhibiting complement-mediated tissue damage, altering B- and T-cell interactions, and suppressing pro-inflammatory cytokines while increasing anti-inflammatory mediators

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Moreover, the Cat8/FacB is responsible for ethanol and acetate utilization and, in consequence, feeding and regulating aspartate, glutamate, TCA and glyoxylate metabolic processes as well as secretion of secondary metabolites 45