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Physiological mediators in nonsteroidal anti-inflammatory drugs (NSAIDs)-induced impairment of gastric mucosal defense and adaptation

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Su propiedad distintiva es la selectividad de receptor en dosis teraputicas de investigacin produce una elevacin mnima de cortisol, prolactina o corticotropina, diferencindolo de GHRPs anteriores como GHRP-6 o GHRP-2

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