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AGEs, in turn, generate ROS through various mechanisms, including metal-catalyzed oxidation reactions

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The main sources of ROI are mitochondria, cytochrome P450 enzymes in the hepatocyte, Kupffer cells and neutrophils [12]

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It attenuates atherosclerosis in ApoE/ mice by reducing bioavailable copper and suppressing vascular inflammation [198] and protects against AAs in ATP7A-deficient mice by inhibiting endothelial ROS [209]