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To support this idea, these studies found that increasing muscle carnitine content saves muscle glycogen during low-intensity exercise (consistent with an increase in muscle lipid utilization), but that increasing muscle carnitine content reduces muscle anaerobic ATP production during high-intensity exercise by better matching glycolytic, pyruvate dehydrogenase complex, and mitochondrial flux [23]

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Following three days of daily injection of phenobarbital or 3-methylcholanthrene, the P450a fluorescence pattern did not change in mature males

Primary carnitine deficiencies are due to defects in carnitine transport into cells or due to defects in synthesis from lysine, although the latter are very rare with only a few cases identified worldwide