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Experimental investigations have reported interactions with VEGFR2 phosphorylation, focal adhesion kinase (FAK) activation, ERK1/2 signaling, nitric oxide-associated pathways, and extracellular matrix regulatory networks

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Furthermore, counteraction occurred with the succinylcholine-induced neuromuscular junction blockade [102], and local anesthesia [103,104] (i.e., via lidocaine intraplantar application and axillary and spinal (L4-L5) intrathecal block [103])