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We postulated that administration of ALC would restore antioxidant levels and improve mitochondrial function, neurotransmission, and neuronal survival in the brains of ethanol-fed mice

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Previous work by our group has indicated that the phenomenon of ROS-induced ROS release can increase the activity of energy-dissipating mitochondrial ion channels, in particular, inner membrane anion channels (IMAC) modulated by mBzR

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