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Compared to the antagonist-bound receptor, the extracellular end of TM7 of these two active receptors laterally move ~5 towards TM6 (measured at C of Q302 7.32 ), presenting a unique TM7 conformational change across class A GPCRs solved to date (Fig

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This immune suppression driven by HE4 allows the cancer to evade immune detection, making it a potential target for therapeutic interventions, particularly in combination with PD-L1/PD-1 checkpoint inhibitors ( Figure 3 ) (92)

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