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Hexosamine pathway Excess glucose not metabolized via glycolysis can enter the hexosamine pathway, which leads to increased production of uridine diphosphate N-acetylglucosamine (UDP-GlcNAc), which is used for O-linked N-acetylglucosamine (O-GlcNAc) modification of proteins

The injection of CagA leads to aberrant cellular signaling, including interactions with Src homology 2 (SH2) domain-containing protein tyrosine phosphatase 2 (SHP2) and phosphoinositide 3-kinase (PI3K), driving the transformation of normal epithelial cells into neoplastic ones (173)

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