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confirmed that a single intranasal dose of Semax in rats produced marked increases in hippocampal BDNF protein and TrkB receptor phosphorylation, alongside improved performance in conditioned avoidance testing establishing BDNF/TrkB modulation as the primary candidate mechanism for Semaxs cognitive research profile and anchoring this compound as a BDNF-upregulating neuropeptide research tool

Govindan, R

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As we get older, our natural production of glutathione begins to decline

Effects of resistance exercise and whey protein supplementation on cognitive function in older men: secondary analysis of a randomised, double-blind, placebo-controlled trial