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We found that overexpression of GmMORF2a or SsCm1 in DD background ( DD/GmMORF2a or DD/SsCm1 ) inhibited cell death and ion leakage after dexamethasone treatment, and inhibited ROS accumulation prior to visible cell death (Fig

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In addition, palbociclib led to the upregulation of genes and proteins involved in lipid metabolism, including glycerophospholipid and triacylglycerol (TAG) biosynthesis, sphingolipid synthesis, arachidonic acid metabolism, and peroxisomal lipid metabolism correlating with the lipidomic findings (Supplemental Fig

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