Genetic invalidation of SLC7A11 has synergistic anti-leukemic activity with APR-246 in vivo We then examined whether inhibition of GSH synthesis through SLC7A11 inhibition could interfere with AML persistence and could enhance APR-246 activity in vivo
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In light of these mixed findings, there is a need for further RCTs specifically assessing these outcomes with adequate follow up as these events may not occur soon after the start of therapy
This enabled identification of 13 C-enrichment in the metabolites originating from the catabolism of 13 C-labelled arginine and proline
