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doi: 10.2147/CMAR.S242531 108 MartignoniE.BlandiniF.GodiL.DesideriS.PacchettiC.ManciniF.et al

Mutations in SLC39A14 disrupt manganese homeostasis and cause childhood-onset parkinsonism-dystonia

In models of hind limb ischemia, it accelerated blood flow recovery and increased vessel density, highlighting its pro-angiogenic properties

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Its a concept of dual targeting, both the environment that the cancer is hosted in and the cancer itself