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This cytokine modulation appears to be a key mechanism underlying its efficacy in models of inflammatory bowel disease, surgical wound healing, and systemic inflammatory injury

[SOURCED] The angiogenic mechanism is closely linked to BPC-157s NO system effects: VEGF-driven angiogenesis is NO-dependent, and the compounds capacity to sustain eNOS activity under stress conditions may create favorable conditions for new vessel formation in ischemic tissue

Morphine is therefore usually given intramuscular (IM) injection to bypass this mechanism

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