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First described in 2000 by the group of Martin Schwab ( While Nogo-A signaling is mainly associated to myelination and inhibition of synaptic and axonal plasticity in adult mammalian CNS, it has also been found to correlate with numerous homeostatic and pathological mechanisms, such as found in schizophrenia ( On animal models for MS, NgR1 expression in B cells has been associated with cell recruitment and inflammation, as well as higher myelin immunoreactivity ( Figure 2