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And because we have these longer-acting medicines than what our own GLP-1 can do, it can last longer

Research indicates the anti-fibrotic effects may be mediated through suppression of transforming growth factor beta (TGF-) signaling, a major driver of pathological cardiac fibrosis, and reduced activation of cardiac fibroblasts

PMID: 60 MartinGRBeckPLSigaletDL

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When GLP-1 medications enable significant weight loss, the reduction in adipose mass directly decreases production of these pro-inflammatory molecules