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Furthermore, the function of SOD2 is concentration dependent: when the intracellular O level exceeds a specific threshold, the activity of SOD2 may exacerbate oxidative damage [22]

DJ-1 protein also contributes to TLR4 receptor endocytosis by palmitoylating targeted lipid rafts to diminish the intensity of its signaling activation (65)

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The latter airs such osteoclastogenic factors, especially of TNF-, IL-1, and IL-18, which eventually uncouple the bone remodeling and feature as the severity of bone loss in older women