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This centrality is underscored by the heart's exceptional dependence on mitochondrial oxidative phosphorylation, which supplies approximately 95% of the ATP required for contraction [23]

Similar to the statin lipid-lowering drugs mentioned earlier, other lipid-lowering drugs, such as probucol, also exhibit the capability to restore the inhibited GSH system and defend the myocardium from oxidative damage after exposure to pathological factors, for example, ISO and cyclophosphamide [242, 243]

It may have neuroprotective benefits that promote neuroplasticity and neuronal survival

Exhaustive genetic profiling studies utilizing the Broad Institute Connectivity Map indicate that the GHK-Cu molecule can actively upregulate or downregulate the expression of over 4,000 distinct human genes