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A pre-clinical study showed that exendin-4 had a remarkable neuroprotective effect, improving the neurological deficit caused by transient middle cerebral artery occlusion in mice, and reducing neuronal loss and microglial inflammation (82)

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In animal models, the co-deletion of LRP5 and LRP6 resulted in severe local and global bone loss in mice (Lim et al., 2021)

A study suggested that in GCH1-deficient mice, BH4 could still accumulate with age, which was associated with the production of BH4 by specific gut microbiota, such as intestinal Actinobacteria [118]

Hendershot, PhD, of the Keck School of Medicine of the University of Southern California, and Klara R