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By knowing the oxidation pathways of UA and detection of its metabolic products, such as, allantoin, hydroxyisourate, triuret, 6-aminouracil, parabanic acid, carboxyimino-6- N -chloroaminopyrimidine-2,4(3 H )-dione (CAPD) and adducts in lysine residues (+140 Da) we may elucidate UA role in specific conditions

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Colorectal cancer (CRC) caused by a number of genetic disorders, including KRAS mutation, p53 mutation, and p53 depletion, which provides additional evidence for the survival-promoting function of p53

Its safety profile has specifically been demonstrated in pediatric populations, where it has been FDA-approved and used without significant adverse outcomes [12, 13]