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A secondary effect of this stress is activation of Nrf2 and its target genes resulting in compensatory higher GSH levels and induction of antioxidant genes in the livers of these mice, which makes these livers more resistant to additional acute stress such as APAP toxicity [151]

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After 24 h, cells were stained with C11-BODIPY, and relative green and red fluorescence was determined (n = 6 technical replicates from 2 independent experiments

Retrieved March 10, 2026, from 7 North Carolina Medicaid, Division of Health Benefits