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This phenomenon, similar to the "shedding phase" observed with minoxidil initiation, may indicate follicles transitioning from telogen (resting) to anagen (growth) phase

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Targeting this system, either by hepatocyte-specific knockout of peroxisomes or by deficiency in ACOX (which catalyzes the initial step in peroxisomal FAO), results in hepatic lipid accumulation and fibrosis, oxidative stress, and inflammation, emphasizing the role of peroximal FAO in NAFLD and NASH [108]