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Glucose-induced membrane depolarization entails opening of voltage-dependent Ca 2+ (VDC) channels, and the resulting Ca 2+ influx then triggers vesicular exocytosis and secretion of GLP-1 into the circulation (Figure 3) [266]

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Chronic GLP-2 treatment does not increase crypt-cell proliferation, and growth of the crypt-villus is reduced in intestinal epithelial-specific IGF knockout mice (Rowland et al., 2011)

Turning that mechanistic insight into medicines is the province of applied discovery, traced in how this receptor biology was turned into a drug-discovery platform

GLP-1 agonists arent likely to cause low blood sugar levels or hypoglycemia when used alone