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Targeting oncometabolism to maximize immunotherapy in Malignant brain tumors

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In the context of brain injury, macrophages exert a pivotal role in the induction of the inflammatory response (Popovich et al., 1999)

In brief, inhibition of TrxR augments the susceptibility of tumor cells to oxidative stress, bolsters the selective eradication of cancerous cells, and instigates the cascade of apoptosis, which has received widespread attention in recent years making TrxR a potential target for cancer therapy (191, 192)