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Meanwhile, the metabolic switch involving mTORC1 inhibition and AMP-activated protein kinase (AMPK) activation in macrophages has been shown to downregulate the aforementioned inflammatory factors, revealing a close link between metabolic signaling pathways and immune inflammation [453], providing dual molecular targets for targeting glycolysis in IBD

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This approach might provide a more informative evaluation of the efficacy of a new therapy by addressing potential confounding factors arising from imbalanced baseline features ( Skewed distributions associated with L-carnitine non-effective patients posed challenges for mean and standard deviation in accurately depicting data tendencies and variability

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