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GHK appears to directly target myofibroblasts, converting pathological persistent collagen deposition to physiological collagen contraction through ITG-1 activation and apoptotic elimination of excess myofibroblasts (Figure 5)

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As we age, the size of the thymus decreases, which some have suggested could be the root cause of age-related decreases to immune function, resulting in higher incidences of infections and autoimmune diseases