Inorganic Chemistry Communications, 140 , 109417
This is exactly what makes the compound so interesting for basic research: if a telomerase-negative cell forms telomerase again through AEDG, elongates its telomeres and keeps dividing, then a boundary assumed to be fundamental is shifted in the laboratory
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But the mechanism of GLP-1 agonists is fundamentally different from AOD 9604, and comparing them directly misses the point
