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Therefore, the pro-apoptotic effect of tamoxifen is partially dependent on the inhibition of FLIP expression and inhibition of pAkt and stimulation of caspase activity in vivo and in vitro, respectively [87]

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The glutamate-induced persistent depolarization affects mitochondrial membrane polarization, impairing mitochondrial function and energy metabolism (see section Oxidative stress and redox imbalance in the CNS, and [91])