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In summary, melatonin could enhance salt stress signaling by up-regulating the expression of genes related to the synthesis of spermine and spermidine in H

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PK/PD simulations The model-predicted mean longitudinal change from baseline profiles in the UPDRS motor scores for SC and SL administration at the doses of 1 mg, 2 mg, 3 mg, and 4 mg, and 20 mg, 30 mg, 40 mg, and 50 mg, respectively, are plotted in Fig

Correspondingly, in OA, p53 signaling may be activated by oxidative stress, DNA damage, or other factors, contributing to cartilage injury (28)