In biohacker forums
The post's table lists macrophage M1/M2 balance as a pathway where M1 activation worsens pain and M2 supports repair, and records the preclinical GLP-1 effect as a shift toward the anti-inflammatory M2 profile
It's important to note that GHK-Cu supports viable follicles but cannot revive completely dead or fibrosed ones
This is why the titration schedule exists
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