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Myeloid-specific blockade of Notch signaling attenuates choroidal neovascularization through compromised macrophage infiltration and polarization in mice

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This finding aligns with data linking hepatic 11HSD1 and brain MAO-A activities in PTSD rats

The formation of gamma-glutamylcysteine is the rate-limiting reaction in GSH synthesis and is feedback inhibited by GSH itself, a mechanism responsible for the regulation of cellular GSH concentration