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Oral NR corrected MASLD phenotypes by decreasing pro-inflammatory mediators, Kupffer cell accumulation, monocyte infiltration, and fibrosis, thereby outperforming SIRT1 overexpression (Zhou et al

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Diese Mechanismen wirken ber einen von den Inkretinen (GLP-1/GIP) getrennten Signalweg und beeinflussen gemeinsam Sttigung, Appetit und Kontrolle des Krpergewichts