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From our point of view, therapeutic options must not only be directed to reach a molecular target, which ultimately would represent a fixed picture of the disease, but also to restore physiological global context in terms of nitro-oxidative stress and inflammatory response, just as the antioxidant treatment seems to act

PMC 3899484

ROS-induced AP-1 prevents the TGF-/Smad signal pathway in fibroblasts that control collagen synthesis to prevent a decrease in collagen production (76)