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Description

Mechanisms include ATP sensitive K + channel closure, facilitation of VDCCs opening, inhibiting membrane repolarization via Kv channels and Ca 2+ -induced Ca 2+ release from cytoplasmic storage sites 9,10,11,12,13,14

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Augments adipocyte energy expenditure and remodels lipid metabolism, with links to polyamine flux and epigenetic marks observed with NNMT knockdown [2]