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13 In this review, we summarise the evidence related to the effects of GLP-1 RAs on these outcomes ( Figure 1 ), which may shed light on the potential of these agents to be used in the clinical care of HF patients and investigated in large clinical trials to evaluate clinical outcomes

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CITED4 inhibits hypoxia-activated transcription in cancer cells, and its cytoplasmic location in breast cancer is associated with elevated expression of tumor cell hypoxia-inducible factor 1

Dulaglutide exploits this property of the Fc fragment to extend the half-life by linking the amino acid-substituted GLP-1 molecule to the N-terminal end of the Fc fragment via a 16-amino acid junction (Figure 2)

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