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Using selected doses and time point for ASA toxicity, we have demonstrated that when GSH synthesis is inhibited in HepG2 cells by buthionine sulfoximine (BSO), prior to ASA treatment, cytotoxicity of the drug is augmented

The relative expression of the target genes was normalized to that of Actb or GAPDH and calculated with the 2Ct method

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Magnesium glycinate and magnesium L-threonate serve different purposes in different tissues

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