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reported that an adult rat was given 0.0625 g/kg of UA before middle cerebral artery closure [128]

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Based on the collective data, we concluded that glutamate inhibited paravascular movement though the NMDA receptor, which was pulsation-dependent, and suppressed tracer penetration into the parenchyma and interstitial fluid drainage though the AMPA/kainate receptor which was AQP4-dependent