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It has been reported that suppression of EMT mediated by histone deacetylase SIRT1 gene silencing or pharmacological inhibition consequently decreases ferroptosis, which further suggests that EMT promotes ferroptosis through epigenetic regulation pathways (Lee J

One potential mechanism is the Nrf2-ARE pathway, which induces a phase 2 detoxification response that promotes disruption of Nrf2-Keap protein interactions, translocation of Nrf2 to the nucleus, and regulation of target gene expression through the ARE, enhancing cellular defense against oxidative damage (Lau et al., 2008)

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