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Graphical Abstract Similar content being viewed by others Introduction Ulcerative colitis (UC), characterized by chronic intestinal inflammation and epithelial barrier dysfunction, remains a therapeutic challenge due to its complex etiology involving dysregulated immune responses, microbiota dysbiosis, and aberrant cell death pathways [1, 2]

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We taper GLP-1 gradually rather than stopping abruptly, and most patients maintain 70-80% of their weight loss long-term when combined with lifestyle integration

One hypothesis: concurrent GIP activation buffers GLP-1-induced nausea

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