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However, favoring the theory of hyperhomocysteinemia-induced platelet activation, in vitro studies have proposed that, besides its direct cytotoxicity that damages endothelial cells (Dudman et al, 1991

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The SMFA is widely used in experiments involving P

These variants often interact with high homocysteine (which upregulates IL-8, MCP-1, and other chemokines via oxidative stress), creating a perfect storm in genetically susceptible individuals