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Huang, S

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Semaglutide reduces appetite via brain GLP-1 receptors, slows gastric emptying, and improves insulin sensitivity

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By integrating engineered bacteria with prebiotics, postbiotics, or conventional drugs in a rational manner, the therapeutic effect can be synergistically enhanced and the multi-factor pathogenesis of liver diseases can be more comprehensively regulated

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Removing one peptide during a cycle while maintaining others can reveal its specific contributions, informing future protocol decisions

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