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There was a notable enrichment of genes resistant to protein-altering variations among genes affected by de novo loss-of-function or protein-altering mutations

doi: 10.3389/fmolb.2024.1440187

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Extensive investigation has elucidated the underlying mechanism, revealing that CagA effectively promotes cholesterol buildup in macrophages by inhibiting the PPAR-LXR pathway, resulting in decreased expression of cholesterol efflux transporters (ABCA1/ABCG1/SR-BI)[55]