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Research shows these microbes may increase anti-osteoclastogenic signals (e.g., IL-10) and decrease inflammatory cytokines (e.g., IL-6, TNF-) that stimulate bone breakdown
On the other hand, treatment of cancer cells with the anti-IGF-IR mAb cixutumumab paradoxically triggers cancer cells to produce IGF2, which in turn recruits macrophages and fibroblasts to promote angiogenesis and metastasis thereby hampering the efficacy of cixutumumab [190]
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