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The expression of SIRT2 is upregulated in the cecal ligation and puncture (CLP)-induced SAKI mouse model and in LPS-treated HK-2 cells, accompanied by significant tubular damage, characterized by the detachment of RTECs, tubular dilation, and epithelial cell flattening

Unlike parenchymal microglia, BAMs act as immune sentinels at CNS borders, interacting with both peripheral and CNS-resident cells

8 Table 1