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Meanwhile, in January 1996 the first rodent study suggested that GLP-1 could be a central regulator of feeding behavior and satiety based on intracerebroventricular injection studies [4], but soon thereafter the results of other rodent studies using peripherally injected GLP-1 argued against an appetite effect [5]

Glutathione also assists in enzymatic reactions and helps other antioxidants, like vitamin E, return to their active form to continue working as antioxidants

Green tea deserves special mention: its catechins (especially EGCG) have been shown to inhibit DPP-IV activity in adipose tissue (view study), potentially preserving GLP-1 levels by slowing its breakdown the same mechanism targeted by DPP-IV inhibitor drugs