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Similar results were found when investigating zebrafish, whereby simple wound healing required only early accumulation of ROS for JNK pathway activation, whilst injuries which necessitated new tissue formation required sustained H 2 O 2 production, activating apoptosis pathways as well as JNK 19,130

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Toxicokinetic analysis revealed that rotigotine behenate-PLGA exhibited sustained release, where Cmax and AUC 0 28 d increased proportionally with the dose with moderate accumulation of rotigotine

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