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TAT-M2PBM, a peptide that disrupts TRPM2-protein kinase C (PKC), a cell-permeable peptide that disrupts TRPM2-PKC coupling, significantly inhibits eNMDARs-mediated excitotoxicity and produces potent protection against ischemic stroke in vitro and in vivo [79]

However, key knowledge gaps remain

Despite the critical role of pericyte-mediated capillary constriction not yet being broadly known in the cardiac community 11 , this finding has been independently reproduced for the heart 12

This suggests potential applications in aging-related liver conditions like non-alcoholic fatty liver disease, where chronic metabolic stress combines with age-associated decline in hepatocyte function