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Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]

De Souza Silva FM, Rocha Alves Da Silva MH, Bracht A, Eller GJ, Constantin RP, Yamamoto NS (2010) Effects of metformin on glucose metabolism of perfused rat livers

Vigorous mixing or agitation can damage peptide structures

PMID: 41978422 Chem Biol Interact Co-exposure to PS-NPs and HFPO-TA potentiates reproductive toxicity via ferroptosis and SLC1A5-Mediated glutamine deprivation in male mice