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Mechanistic studies reveal that JMJD3 facilitates TGF-/Smad signaling and epithelialmesenchymal transition (EMT) by regulating syntenin, a scaffolding protein that enhances TGF- signaling

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MST is a TMS variant with a more focused superficial field and minimal stimulation of inner brain structures such as hippocampus (127), thus having lower incidences of cognitive deficits unlike ECT (128) but with antidepressant effect similar to RUL ECT (129, 130)