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In models of NOS inhibitor-induced hypertension, BPC-157 attenuated blood pressure changes, suggesting interaction with the NO pathway rather than simple NO donation

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In another migration/scratch wound assay, BPC-157 significantly increased the open wound area, suggesting that the motility of endothelial cells across wounds was improved

BPC 157 Rescued NSAID-Cytotoxicity Via Stabilizing Intestinal Permeability and Enhancing Cytoprotection Current Pharmaceutical Design BPC-157 counteracted NSAID-induced intestinal damage by stabilizing intestinal permeability, restoring cytoprotective prostaglandin systems, and recovering leaky-gut syndrome pathways